EDGE Los Angeles

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  Anonymous, 2012-04-30 16:46:58

  Acording to my opinion; what is the common denominator of the neurodegenerative diseases (mad cow disease...) , including Alzheimer’s disease? This was 11 years ago ( March 2001), when I published (in Czech) an alternative theory ( BSE ammonia- magnesium theory), where the main role of NMDA receptors was described. In addition, in scientific literature there was described the effect of drugs, in Alzheimer’s disease in humans, on the principle of control hyperfunction of NMDA receptors, which is consistent with my BSE alternative theory of BSE. See more about the Nameda; A medication known as Namenda® (memantine), an N-methyl D-aspartate (NMDA) antagonist, is prescribed to treat moderate to severe Alzheimer’s disease (www.nia.nih.gov/alzheimers/publication/alzheimers-disease-medications-fact-sheet I worked in the USA almost one year- West Virginia University (1991, Morgantown). There I obtained a lot of information from scientific journals (in former Czechoslovakia it was not possible), what I used to create an alternative theory of the origin and the spread of mad cow disease (BSE). Relevant findings I have published 11 years ago in Czech (March 2001) and in English (May 2002; Netherlands, International Journal "Feed Mix"; www.warmwell.com/lone_voices_in_the_bse_debate[1].pdf ). Later, in August 2006, I published own website, see; www.bse-expert.cz Since then I pointed my website in the order of hundreds of magazines around the world, especially in the U.S.. See some of them, for example on Google; www.google.com/search?q=hlasny+bse+expert&hl=cs&lr=&start=0&sa=N&filter=0 . In 2008, I repeatedly visited the USA, it was about the occasion of my presentation in Vancouver, Canada (July 2008; 29th World Veterinary Congress; Neurodegenerative Diseases and Schizophrenia as a Hyper or Hypofunction of the NMDA Receptors (www.bse-expert.cz/pdf/Veter_kongres.pdf). According to my theory, the origins of the neurodegenerative diseases may lie in chronic magnesium deficiency coupled with a high protein intake. So defective prions are markers of the diseases rather than the cause and BSE can be a naturally occurring disease, not an infectious disease. WHY? Because, about the BSE/ vCJD diseases; this was never justified scientifically! It was pure, math-model-driven science fiction. But it was pushed very vigorously by the British science establishment, which has never confessed to its errors... See more about the; BSE/ vCJD mathematical- models, see recent large three comments (February 2010) in Telegraph.co.uk (www.telegraph.co.uk/health/healthnews/7168326/Does-vCJD-still-pose-a-major-public-health-threat.html ). However, well-known circumstances can be show (as a detective story); in documenting the first case of disease transmission, by blood transfusion. For more informations see my other large comments (January 12, 2011) in Western Star (www.thewesternstar.com/News/Canada%20-%20World/Business/1969-12-31/article-2095060/Womans-death-in-northern-Italy-is-nations-2nd-fatal-case-of-mad-cow-disease/1 ). At the present time I will complete my website to the final version, so it would be clear among other things, that mad cow disease has never been and is not an infectious disease. Among other things, there much contributed recent research about the Alzhemeir´s disease.